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References

  1. Simsek S et al. Rapid Rh D genotyping by polymerase chain reaction-based amplification of DNA. Blood, 1995. [Citation] [RHeference]
  2. Beckers EA et al. The R0Har RH:33 phenotype results from substitution of exon 5 of the RHCE gene by the corresponding exon of the RHD gene. Br J Haematol, 1996. [Citation] [RHeference]
  3. Beckers EA et al. The RoHar antigenic complex is associated with a limited number of D epitopes and alloanti-D production: a study of three unrelated persons and their families. Transfusion, 1996. [Citation] [RHeference]
  4. Faas BH et al. Involvement of Ser103 of the Rh polypeptides in G epitope formation. Transfusion, 1996. [Citation] [RHeference]
  5. Beckers EA et al. The genetic basis of a new partial D antigen: DDBT. Br J Haematol, 1996. [Citation] [RHeference]
  6. Beckers EA et al. Characterization of the hybrid RHD gene leading to the partial D category IIIc phenotype. Transfusion, 1996. [Citation] [RHeference]
  7. Faas BH et al. Molecular background of VS and weak C expression in blacks. Transfusion, 1997. [Citation] [RHeference]
  8. Maaskant-van Wijk PA et al. Evidence That the RHDVI Deletion Genotype Does Not Exist Blood, 1997. [Citation] [RHeference]
  9. Daniels GL et al. The VS and V blood group polymorphisms in Africans: a serologic and molecular analysis. Transfusion, 1998. [Citation] [RHeference]
  10. Maaskant-van Wijk PA et al. Genotyping of RHD by multiplex polymerase chain reaction analysis of six RHD-specific exons. Transfusion, 1998. [Citation] [RHeference]
  11. Flegel WA et al. In-frame triplet deletions in RHD alter the D antigen phenotype. Transfusion, 2006. [Citation] [RHeference]
  12. von Zabern I et al. IVS5-38del4 deletion in the RHD gene does not cause a DEL phenotype: relevance for RHD alleles including DFR-3. Transfusion, 2007. [Citation] [RHeference]
  13. Flegel WA et al. DCS-1, DCS-2, and DFV share amino acid substitutions at the extracellular RhD protein vestibule. Transfusion, 2008. [Citation] [RHeference]
  14. Flegel WA et al. D variants at the RhD vestibule in the weak D type 4 and Eurasian D clusters. Transfusion, 2009. [Citation] [RHeference]
  15. Flegel WA et al. Six years' experience performing RHD genotyping to confirm D- red blood cell units in Germany for preventing anti-D immunizations. Transfusion, 2009. [Citation] [RHeference]
  16. Schmid P et al. Specific amino acid substitutions cause distinct expression of JAL (RH48) and JAHK (RH53) antigens in RhCE and not in RhD. Transfusion, 2010. [Citation] [RHeference]
  17. von Zabern I et al. DFR-5 is a New Member in the DFR Family of Partial D and Has the Hybrid Structure RHD-CE(3-4)-D Transfus Med Hemother, 2012. — Abstract — [RHeference]
  18. von Zabern I et al. D category IV: a group of clinically relevant and phylogenetically diverse partial D. Transfusion, 2013. [Citation] [RHeference]
  19. C von Arx et al. A novel RHD splice donor site mutation leading to a RhD-negative phenotype Vox Sanguinis, 2019. — Abstract — [RHeference]