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References

  1. Le Pennec PY et al. A serologic study of red cells and sera from 18 Rh:32,-46 (RN/RN) persons. Transfusion, 1989. [Citation] [RHeference]
  2. Legler TJ et al. RHD genotyping in weak D phenotypes by multiple polymerase chain reactions. Transfusion, 1998. [Citation] [RHeference]
  3. Hyodo H et al. New RhD(IVb) identified in Japanese. Vox Sang, 2000. [Citation] [RHeference]
  4. Hyodo H et al. Polymorphisms of RhD(Va) and a new RhD(Va)-like variant found in Japanese individuals. Vox Sang, 2000. [Citation] [RHeference]
  5. Legler TJ et al. D(Va) category phenotype and genotype in Japanese families. Vox Sang, 2000. [Citation] [RHeference]
  6. Legler TJ et al. RHD sequencing: a new tool for decision making on transfusion therapy and provision of Rh prophylaxis. Transfus Med, 2001. [Citation] [RHeference]
  7. Shao CP et al. Molecular background of Rh D-positive, D-negative, D(el) and weak D phenotypes in Chinese. Vox Sang, 2002. [Citation] [RHeference]
  8. Noizat-Pirenne F et al. Rare RHCE phenotypes in black individuals of Afro-Caribbean origin: identification and transfusion safety. Blood, 2002. [Citation] [RHeference]
  9. Wagner FF et al. RHD allele distribution in Africans of Mali. BMC Genet, 2003. [Citation] [RHeference]
  10. Körmöczi GF et al. Molecular and serologic characterization of DWI, a novel "high-grade" partial D. Transfusion, 2004. [Citation] [RHeference]
  11. Wagner T et al. Anti-D immunization by DEL red blood cells. Transfusion, 2005. [Citation] [RHeference]
  12. Körmöczi GF et al. Novel weak D types 31 and 32: adsorption-elution-supported D antigen analysis and comparison to prevalent weak D types. Transfusion, 2005. [Citation] [RHeference]
  13. Körmöczi GF et al. A comprehensive analysis of DEL types: partial DEL individuals are prone to anti-D alloimmunization. Transfusion, 2005. [Citation] [RHeference]
  14. Gassner C et al. Presence of RHD in serologically D-, C/E+ individuals: a European multicenter study. Transfusion, 2005. [Citation] [RHeference]
  15. Flegel WA et al. DCS-1, DCS-2, and DFV share amino acid substitutions at the extracellular RhD protein vestibule. Transfusion, 2008. [Citation] [RHeference]
  16. Müller SP et al. The determination of the fetal D status from maternal plasma for decision making on Rh prophylaxis is feasible. Transfusion, 2008. [Citation] [RHeference]
  17. Yukari Nishiyama et al. A case of suspected partial D because of weak reactivity to anti-D on column agglutination technology and identified as partial D (DBT-1) by genetic testing Japanese Journal of Transfusion and Cell Therapy, 2011. — Article — [RHeference]
  18. Dogic V et al. Distribution of weak D types in the Croatian population. Transfus Med, 2011. [Citation] [RHeference]
  19. Krog GR et al. Is current serologic RhD typing of blood donors sufficient for avoiding immunization of recipients? Transfusion, 2011. [Citation] [RHeference]
  20. Orzińska A et al. RHD variants in Polish blood donors routinely typed as D-. Transfusion, 2013. [Citation] [RHeference]
  21. Gassner C et al. Novel RHD alleles with weak hemagglutination and genetic Exon 9 diversity: weak D Types 45.1, 75, and 76. Transfusion, 2013. [Citation] [RHeference]
  22. Jakobsen MA et al. A case of high-titer anti-D hemolytic disease of the newborn in which late onset and mild course is associated with the D variant, RHD-CE(9)-D. Transfusion, 2014. [Citation] [RHeference]
  23. Van Sandt VS et al. RHD variants in Flanders, Belgium. Transfusion, 2015. [Citation] [RHeference]
  24. Ogasawara K et al. Molecular basis for D- Japanese: identification of novel DEL and D- alleles. Vox Sang, 2015. [Citation] [RHeference]
  25. Clarke G et al. Resolving variable maternal D typing using serology and genotyping in selected prenatal patients. Transfusion, 2016. [Citation] [RHeference]
  26. Ogasawara K et al. Weak D alleles in Japanese: a c.960G>A silent mutation in exon 7 of the RHD gene that affects D expression. Vox Sang, 2016. [Citation] [RHeference]
  27. Turley E et al. Severe hemolytic disease of the fetus and newborn due to allo-anti-D in a patient with a partial DEL phenotype arising from the variant allele described as RHD*148+1T (RHD*01EL.31). Transfusion, 2018. [Citation] [RHeference]
  28. Srivastava K et al. Two distinct RHCE alleles in cis to weak D type 31 alleles in individuals from different ethnicities. Transfusion, 2018. [Citation] [RHeference]
  29. Kim H et al. Reconsidering RhD positive blood transfusion for Asia type DEL patients. Transfus Apher Sci, 2019. [Citation] [RHeference]
  30. M Pisacka et al. Strong pregnancy induced anti-D immunization in del phenotype with RHD*01EL.04 allele Vox Sanguinis, 2019. — Abstract — [RHeference]
  31. Koutsouri T et al. Frequency distribution of RHD alleles among Greek donors with weak D phenotypes demonstrates a distinct pattern in central European countries. Transfus Med, 2019. [Citation] [RHeference]
  32. Matzhold EM et al. RHD del28Phe (DMW) encoded by a novel in-frame deletion resulting in reduced D antigen expression. Transfusion, 2019. [Citation] [RHeference]
  33. Guzijan G et al. Implementation of Molecular RHD Typing at Two Blood Transfusion Institutes from Southeastern Europe. Transfus Med Hemother, 2019. [Citation] [RHeference]
  34. Vege S et al. Impact of RHD genotyping on transfusion practice in Denmark and the United States and identification of novel RHD alleles. Transfusion, 2021. [Citation] [RHeference]