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References

  1. Colin Y et al. Genetic basis of the RhD-positive and RhD-negative blood group polymorphism as determined by Southern analysis. Blood, 1991. [Citation] [RHeference]
  2. Le van Kim C et al. Molecular cloning and primary structure of the human blood group RhD polypeptide. Proc Natl Acad Sci U S A, 1992. [Citation] [RHeference]
  3. Chérif-Zahar B et al. Molecular analysis of the structure and expression of the RH locus in individuals with D--, Dc-, and DCw- gene complexes. Blood, 1994. [Citation] [RHeference]
  4. Mouro I et al. Rearrangements of the blood group RhD gene associated with the DVI category phenotype. Blood, 1994. [Citation] [RHeference]
  5. Rouillac C et al. Transcript analysis of D category phenotypes predicts hybrid Rh D-CE-D proteins associated with alteration of D epitopes. Blood, 1995. [Citation] [RHeference]
  6. Rouillac C et al. Lack of G blood group antigen in DIIIb erythrocytes is associated with segmental DNA exchange between RH genes. Br J Haematol, 1995. [Citation] [RHeference]
  7. Rouillac C et al. Leu110Pro substitution in the RhD polypeptide is responsible for the DVII category blood group phenotype. Am J Hematol, 1995. [Citation] [RHeference]
  8. Cartron JP et al. Tentative model for the mapping of D epitopes on the RhD polypeptide. Transfus Clin Biol, 1996. [Citation] [RHeference]
  9. Noizat-Pirenne F et al. Molecular analysis of selected Rh variants. Transfus Clin Biol, 1996. [Citation] [RHeference]
  10. Rouillac C et al. Molecular basis of the altered antigenic expression of RhD in weak D(Du) and RhC/e in RN phenotypes. Blood, 1996. [Citation] [RHeference]
  11. Matassi G et al. Characterization of the recombination hot spot involved in the genomic rearrangement leading to the hybrid D-CE-D gene in the D(VI) phenotype. Am J Hum Genet, 1997. [Citation] [RHeference]
  12. Maaskant-van Wijk PA et al. Evidence That the RHDVI Deletion Genotype Does Not Exist Blood, 1997. [Citation] [RHeference]
  13. Chérif-Zahar B et al. Shift from Rh-positive to Rh-negative phenotype caused by a somatic mutation within the RHD gene in a patient with chronic myelocytic leukaemia. Br J Haematol, 1998. [Citation] [RHeference]
  14. Noizat-Pirenne F et al. DAL: a new partial RHD phenotype. Transfusion, 2001. [Citation] [RHeference]
  15. Noizat-Pirenne F et al. Rare RHCE phenotypes in black individuals of Afro-Caribbean origin: identification and transfusion safety. Blood, 2002. [Citation] [RHeference]
  16. Pham BN et al. Heterogeneous molecular background of the weak C, VS+, hr B-, Hr B- phenotype in black persons. Transfusion, 2009. [Citation] [RHeference]